The true statement about contrast induced acute kidney injury is:
Iodinated contrast agents are a leading cause of AKI. Increased risk is seen in Chronic kidney disease - paicularly diabetic nephropathy The most common clinical course of contrast nephropathy is A rise in SCr beginning 24-48 hours following exposure Peaking within 3-5 days Resolving within 1 week ALSO NOTE: More severe, dialysis-requiring AKI is uncommon Patients with multiple myeloma and renal disease are paicularly susceptible for severe renal failure. Low fractional excretion of sodium and without features of tubular necrosis are common findings. Ref: Harrison-E-18, P-2297.
Rise in the serum creatinine after 12 hours
Peak in serum creatinine in 2 weeks
Dialysis requirement is usually uncommon
High fractional excretion of Na is a common finding