All of the following statements about acute adrenal insufficiency are true, EXCEPT:
Adrenal insufficiency is associated with decreased levels of glucocoicoids, mineralocoicoids and androgens. Normally coisol is involved in hepatic glycogen synthesis, gluconeogenesis and release of glucose into the circulation. So in adrenal insufficiency, deficiency of coisol results in hypoglycemia. Adrenal insufficiency can be primary (resulting from adrenal disease), or secondary (due to a deficiency of ACTH). Primary adrenal insufficiency can be caused by autoimmune disease, infections, and metastatic deposits. Most common cause of secondary adrenal insufficiency is therapy with exogenous glucocoicoids. Ref: Schwaz's Principles of Surgery, 9th Edition, Chapter 38; Modern Nutrition In Health And Disease By Maurice Edward Shils, 10th Edition, Page 646; Davidson's Principles and Practice of Medicine, 21st Edition, Page 775, Box: 20.45
Hyperglycemia is usually present
Acute adrenal insufficiency usually is secondary to exogenous glucocoicoid administration
Acute adrenal insufficiency presents with weakness, vomiting, fever, and hypotension
Hyponatremia occurs because of impaired renal tubule sodium resorption