The exact pathogenesis of<unk> -alkali syndrome is unknown. Hypercalcemia in<unk> -alkali syndrome involves several mechanisms including: intestinal absorption of calcium is increased, bone buffering of calcium becomes saturated, and renal excretion of calcium is decreased. Several factors that increase bicarbonate reabsorption and contribute to the alkalosis in<unk> -alkali syndrome include: volume depletion due to increased sodium and free water excretion caused by increased calcium intake, suppression of PTH, direct tubular effects of calcium and other factors that cause volume depletion or alkalosis such as vomiting or thiazide use.
Milk-alkali syndrome pathophysiology
Milk-alkali syndrome risk factors
Acute diarrhea pathophysiology
Ileus risk factors
Meckel's diverticulum pathophysiology
Milk-alkali syndrome differential diagnosis
Lactic acidosis pathophysiology
Ileus pathophysiology