<unk>  syncope is defined as a syndrome in which triggering of a neural reflex results in a usually self-limited episode of systemic hypotension characterized by both bradycardia (asystole or relative bradycardia) and peripheral vasodilation.
It is caused by an abnormal or exaggerated autonomic response to various stimuli, commonly standing and emotion. The underlying mechanism for this exaggerate autonomic response remains unclear; however, the activation of cardiac C fibres has shown to play role which subsequently causes reflex-mediated changes in the vascular tone and heart rate. On the contrary, failure of autonomic reflex response results in orthostatic hypotension.
The pathogenesis for syncope in patients with hypersensitive carotid sinus and<unk>  syncope remain the same with the only difference being the receptor involved to stimulate the brainstem nucleus.
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