Thyroid<unk> s may arise from different cells in the thyroid parenchyma. The pathogenesis of developing a thyroid<unk>  may differ based on the type of the<unk> , and whether it is malignant or benign. Basically thyroid<unk> s may develop secondary to hyperplasia, mutations and resultant carcinoma, excess colloid accumulation, or frominflammation of thyroid tissue. Genetic mutation is considered as one of the most important mechanisms of developing thyroid<unk> s, especially neoplastic thyroid<unk> s. Most of these mutations occur as somatic mutations, while some may exhibit familial inheritance. The most important variety of familial thyroid cancers are caused by genetic mutations, and are called familial non-medullary thyroid cancer (FNMTC). Other important genes related to thyroid<unk>  formation include, N&H ras, RET, Gsp, C-MET, TRK, EGF / EGF-R, and P53.
Thyroid nodule pathophysiology
Thyroid adenoma pathophysiology
Medullary thyroid cancer pathophysiology
Thyroid adenoma classification
Thyroid adenoma causes
Papillary thyroid cancer risk factors
Parathyroid adenoma pathophysiology
Medullary thyroid cancer risk factors