<unk>  usually appears within minutes to hours after a cerebral injury. It is an acute life-threatening complication associated with many forms of central nervous system injury, such as brain or spinal cord hemorrhage, trauma, tumors, epilepsy and infections. The pathogenetic factors for the its onset include increased intracranial pressure and severe over-activation of the sympathetic nervous system.<unk>  must be differentiated from other diseases with same symptoms, include aspiration pneumonia, cardiogenic<unk>  edema, angioedema and asthma attack.<unk>  is characterized by dyspnea, bilateral basal<unk>  crackles and the other signs and symptoms of<unk>  edema, in the absence of cardiac failure. Arterial blood gas test shows hypoxemia, hypercapnia and acidosis. In treatment of<unk> , the main principle is supportive treatment and decreasing intracranial pressure as in acute respiratory distress syndrome.
Neurogenic pulmonary edema
Decompression sickness
Cyclical edema syndrome
Fat embolism syndrome
Capillary leak syndrome
Adams-Stokes syndrome
Cholesterol emboli syndrome
COACH syndrome