<unk>  is the formation of non-solid feces. Stools may also float due to excess fat from malabsorption, have an oily appearance and be foul smelling. An oily anal leakage or some level of fecal incontinence may occur. There is increased fat excretion, which can be measured by determining the fecal fat level. While definitions have not been standardized, fat excretion in feces in excess of 0.3 (g/kg) / day is considered indicative of<unk> . Based on underlying etiology<unk>  can be classified into 3 types, intestinal, biliary, and pancreatic<unk> .<unk>  occurs as a result of either defect of the normal architecture of digestive tract or defect of synthesis or secretion of enzymes required for metabolism fats.<unk>  may be caused by Celiac disease, choledocholithiasis, cystic fibrosis, exocrine pancreatic insufficiency, hypolipidemic drugs, inflammatory bowel disease, small bowel bacterial overgrowth syndrome. Common risk factors in the development of<unk>  include Celiac disease, cystic fibrosis, exocrine pancreatic insufficiency, inflammatory bowel disease, small intestinal bacterial overgrowth, hypolipidemic drugs. If left untreated,<unk>  can lead to severe malnutrition due to inability of gastrointestinal tract to absorb fat soluble vitamins and ultimately severe weight loss. Complication of<unk>  include anemia, intestinal obstruction, weight loss. Prognosis of<unk>  is generally good with appropriate treatment. The 72 hr-fecal fat determination is the gold standard test for the diagnosis of<unk> . Mild<unk>  can manifest as passage of frothy, foul smelling stool, greasy in appearance and difficult to flush, abdominal pain, bloating, heart burn. if severe it may cause malnutrition , dehydration, anemia, muscle weakness, weight loss, skin problems, neurological problems, osteoporosis. Patients with<unk>  usually appear emaciated secondary to loss of subcutaneous fat. Physical examination of patients with<unk>  is usually remarkable for distended abdomen, orthostatic hypo-tension and ecchymoses, Chvostek sign and Trousseau sign secondary to hypocalcemia. There are no specific laboratory findings associated with<unk> . Management of<unk>  include treatment of underlying etiology, control of diarrhea and correction of nutritional deficiencies. Surgery is usually reserved for patients with refractory or pre-malignant complications, such as Enteropathy Associated T-cell Lymphoma (EATL) and ulcerative jejunitis (UJ).  Effective measures for the primary prevention of<unk>  include smoking cessation, alcohol cessation, minimizing the use of certain medications, such as antibiotics, that can alter normal bowel flora, and consuming diet rich in dietary fiber.
Steatorrhea
Aspermia
Galactorrhea
Constipation  
Constipation
Achlorhydria
Morphea
Drooling