During either an infection or a hypersensitivity reaction, the inflammatory response may cause myonecrosis either directly or indirectly as part of an autoimmune reaction. Histopathological features include abundant edema in the myocardial interstitium and an inflammatory infiltrate which is rich in lymphocytes and macrophages.  Focal destruction of myocytes as a result of the inflammatory process results in left ventricular dysfunction.
Myocarditis pathophysiology
Endocarditis pathophysiology
Endocarditis diagnostic criteria
Sinoatrial arrest pathophysiology
Pacemaker syndrome pathophysiology
Ventricular remodeling pathophysiology
Uremic pericarditis pathophysiology
Tuberculous pericarditis pathophysiology