<unk>  refers to the changes in size, shape, and function of the heart after injury to the left ventricle.  The injury is typically due to acute myocardial infarction (usually transmural or ST segment elevation infarction), but may be from a number of causes that result in increased pressure or volume overload (forms of strain) on the heart. Chronic hypertension, congenital heart disease with intracardiac shunting, and valvular heart disease may also lead to<unk> . After the insult occurs, a series of histopathological and structural changes occur in the left<unk>  myocardium that lead to progressive decline in left<unk>  performance.  Ultimately,<unk>  may result in diminished contractile (systolic) function and reduced stroke volume.
Medically speaking,<unk> " implies a decline in function (even though the word<unk> " usually implies improvement). The term "reverse<unk> " in cardiology implies an improvement in<unk>  mechanics and function after a remote injury.
Ventricular remodeling
Cardiac fibrosis
Myocardial inflammation
Myocardial edema
Coronary vasospasm
Ventricular hypertrophy
Digoxin effect
Myocardial disarray