<unk>  <unk>  is an autosomal dominant classical<unk>  subtype that typically includes hemiparesis (weakness of half the body) during the aura phase. It can be accompanied by other symptoms, such as ataxia, coma and epileptic seizures. There is clinical overlap in some<unk>  patients with episodic ataxia type 2 and spinocerebellar ataxia type 6, benign<unk>  infantile convulsions, and alternating<unk>  of childhood. There are 3 known loci for<unk> .<unk> 1, which accounts for approximately 50% of<unk>  patients, is caused by mutations in a gene coding for the P/Q-type calcium channel α subunit, CACNA1A.<unk> 1 is also associated with cerebellar degeneration.<unk> 2, which accounts for <25% of<unk>  cases, is caused by mutations in the Na+/K+-ATPase gene ATP1A2.<unk> 3 is a rare subtype of<unk>  and is caused by mutations in a sodium channel α-subunit coding gene, SCNA1. These three subtypes do not account for all cases of<unk> , suggesting the existence of at least one other locus <unk> 4). Many of the non-familial cases of<unk><unk>  (sporadic<unk><unk>  are also caused by mutations at these loci.
Familial hemiplegic migraine
Cluster headache
Red wine headache
Trigeminal neuralgia
Benign positional vertigo 
Benign paroxysmal positional vertigo
Migraine
Transient ischemic attack