At present, the pathophysiology of non-atherosclerotic spontaneous coronary<unk><unk>  (NA-SCAD) continues to be poorly understood due to the rarity of this condition and its heterogeneous pathology. Although intimal tear or bleeding of vasa vasorum with intermedial hemorrhage seems to be the most probable reason, the exact underlying mechanism is still unknown. In a SCAD registry, a total of 5% to 8% of patients were carriers of genetic mutations for connective tissue disorders. SCAD may also be associated with a variety of disorders including but not limited to fibromuscular dysplasia, Connective tissue disorders, and autoimmune diseases such as systemic lupus erythematous.
Spontaneous coronary artery dissection pathophysiology
Spontaneous coronary artery dissection epidemiology and demographics
Spontaneous coronary artery dissection other imaging findings
Papillary muscle rupture pathophysiology
ST elevation myocardial infarction pathophysiology
Spontaneous coronary artery dissection laboratory findings
Spontaneous coronary artery dissection treatment approach
Spontaneous coronary artery dissection diagnostic approach