<unk>  vein thrombosis (DVT) results from the formation of a blood clot in the<unk>  veins.  Three mechanisms predispose to DVT, they are collectively described as the Virchow's triad. 1. Alterations in blood flow (stasis): Venous stasis is a major risk factor for the development of thrombosis. It occurs in certain pathological conditions (as in heart failure) wherein it causes an increase in platelet to endothelium contact and decreases the dilution of clotting factors. This increases the risk of clot formation, and it forms microthrombi, which further grow and propagate. 2. Injury to the vascular endothelium (Endothelial dysfunction): Intrinsic or secondary to external trauma, such as catheterization, can cause intimal damage and stimulate clot formation. 3. Alterations in the constitution of blood (Hypercoagulability): Abnormal changes in coagulation can increase the propensity to develop thrombosis.
Deep vein thrombosis pathophysiology
Portal vein thrombosis pathophysiology
Varicose veins pathophysiology
Thrombosis pathophysiology
Varicose veins risk factors
Deep vein thrombosis risk factors
Suppurative thrombophlebitis risk factors
Stent thrombosis risk factors