The pathogenesis of<unk>  arthritis  involves prominent T-lymphocytic infiltrate, particularly CD4 cells in the skin and joints. High levels of tumor necrosis factor alpha (TNF), IL-8, IL-6, IL-1, IL-10, and matrix metalloproteinases are present in the synovial fluid of patients with early<unk>  arthritis.The elevated levels of TNF and various interleukins lead to a high number of osteoclast precursor cells circulating in the blood which ultimately leads to joint destruction.
Psoriatic arthritis pathophysiology
Rheumatoid arthritis pathophysiology
Septic arthritis pathophysiology
Ankylosing spondylitis pathophysiology
Bursitis pathophysiology
Osteoarthritis pathophysiology
Systemic lupus erythematosus pathophysiology
Rheumatic fever pathophysiology