<unk> s form when the intraventricular tension stretches the non contracting, infarcted heart muscle, resulting in an expansion of the thin layer of necrotic muscle and fibrous tissue, which bulges with each cardiac contraction. The wall of a mature<unk>  is a white fibrous scar. It becomes more densely fibrotic as the time passes, and bulges outward with each cardiac contraction, resulting in a reduction of the left ventricular stroke volume. On microscopy, hyalinized fibrous tissue is the predominant finding. It usually takes 1 month for fibrous tissue to form.
Left ventricular aneurysm pathophysiology
Aortic regurgitation pathophysiology
Aortic stenosis pathophysiology
Thoracic aortic aneurysm pathophysiology
Tricuspid stenosis pathophysiology
Aortic dissection pathophysiology
Tricuspid regurgitation pathophysiology
Left ventricular aneurysm differential diagnosis