<unk>  is a clinical scenario of mineralocorticoid excess with resistant hypertension, hypokalemia, and metabolic alkalosis due to increased hydrogen ion excretion. Aldosteronism may be classified into three types, primary<unk>  (Conn's syndrome), secondary<unk> , and pseudohyperaldosteronism. Primary<unk>  can caused by aldosterone-secreting adenoma, bilateral hyperplasia of the adrenal glands, and ectopic secretion of aldosterone from ovaries and kidneys. Primary<unk>  features overproduction of aldosterone despite suppressed plasma renin activity (PRA). The resulting Na+ retention produces hypertension, and increased K+ excretion may cause hypokalemia. Secondary<unk>  is caused by high renin and subsequently aldosterone level, such as renovascular causes and reninoma. Pseudohyperaldosteronism is the clinical presentation of<unk>  such as resistant hypertension, hypokalemia, and metabolic alkalosis due to factors other than renin and  aldosterone. The treatment should be prescribed for the blockade of aldosterone effects, or based on underlying disease.
Hyperaldosteronism
Secondary hyperaldosteronism
Familial hypoaldosteronism
Hypoaldosteronism
Primary hyperaldosteronism
Acromegaly
Familial hyperreninemic hypoaldosteronism type 2
Thyromegaly