<unk>  T<unk><unk>  arises from an immature cytotoxic T-cell clonally expressing the γδ T-cell receptor. Gamma delta (γδ) T<unk> s constitute 1% to 5% of the circulating lymphocytes in human blood. These<unk> s preferentially home in on some epithelial rich tissues and sinusoidal areas of the splenic red pulp where they represent up to 30% of the whole T-cell population.<unk>  γδ T-cell<unk>  (HSTCL) is a rare aggressive subtype of extra nodal<unk> . Genes involved in the pathogenesis of<unk>  T<unk><unk>  include isochromosome 7q, TRG@, and T-cell receptor (TCR) Gamma/Delta genes. On gross pathology, diffuse enlargment of liver and spleen are characteristic findings of<unk>  T<unk><unk> . On microscopic histopathological analysis, monotonous appearance with a small amount of cytoplasm and inconspicuous nucleoli are characteristic findings of neoplastic<unk> s of<unk>  T<unk><unk> .
Hepatosplenic T cell lymphoma pathophysiology
Autoimmune hepatitis pathophysiology
Amoebic liver abscess pathophysiology
Drug induced liver injury pathophysiology
Alcoholic hepatitis pathophysiology
Pyogenic liver abscess pathophysiology
Hepatocellular carcinoma pathophysiology
Amoebic liver abscess epidemiology and demographics