Like many forms of glomerulopathies,<unk>  glomerulonephritis (glomerulopathy) has been a diagnosis of tissue pathology rather the diagnosis of a specific disease entity. Therefore the term membranoploriferative glomerulonephritis (MPGN) relates to a pattern of glomerular injury which can be caused by many disease states. Historically the nephropathologists divided MPGN into 3 distinctive categories to shed light into what may be causing this type of kidney injury: 
MPGN type 1: mesangial and subendothelial electron dense deposits MPGN type 2: electron dense material in the glomerular basement membrane
MPGN type 3: subepithelial deposits with basement membrane spikes This categorization, however is now out of date. The recognition of several new disorders as the underlying cause of MPGN, and the lack of clinical, prognostic or therapeutic relevance made this categorization less useful. For completeness and to help better accommodate the transition from old to new<unk> , below both<unk> s are reviewed.<unk>  of MPGN based on immunofluorescence microscopy is a result of all advances in the understanding of the pathogenesis of the disease. Based on this advanced techniques, there are three types of MPGN :Immune-complex-mediated MPGN (Type I) ,Complement-mediated MPGN  (Type II), Non-Ig/complement-mediated MPGN  (Type III)
Membranoproliferative glomerulonephritis classification
Rapidly progressive glomerulonephritis classification
Microscopic polyangiitis classification
Interstitial nephritis epidemiology and demographics
C4 glomerulopathy Medical therapy
Membranous glomerulonephritis laboratory findings
KDIGO guidelines classification scheme
Cryoglobulinemia classification