Pathogenensis of<unk>  meningitis may include the transmission, colonization, invasion and seeding of meninges. It is a complex process involving interaction between<unk>  pathogenic elements and host immune response. There may be genetic predisposition to develop infection. The sequence of events that may ensue after<unk>  invasion of meninges include injury to the blood brain or blood CSF barrier, disruption of intercellular tight junctions, vasogenic edema, loss of cerebral autoregulation, increased intra cranial pressure, and development of Signs and symptoms due to raised IC pressure. The inflammatory molecules which may play a role in this pathogenic process includes interleukins IL1, IL6, TNF and matrix metalloproteinases.
Bacterial meningitis pathophysiology
Viral meningitis pathophysiology
Neoplastic meningitis pathophysiology
Fungal meningitis pathophysiology
Viral encephalitis pathophysiology
Viral encephalitis causes
Viral meningitis risk factors
Viral encephalitis risk factors