The pathogenesis of infective endocarditis includes valvular damage, altered and turbulent flow, bacteremia, and lack of blood supply to the valves. Damaged endothelium becomes a site for attachment of infectious agents in infectious endocarditis.  Nonbacterial thrombotic endocarditis is related to hypercoagulable states such as pregnancy or systemic bacterial infection. The characteristic lesion of endocarditis is vegetation.  Vegetations are composed of fibrin, inflammatory cells, platelets, and microorganisms.
Endocarditis pathophysiology
Myocarditis pathophysiology
Endocarditis diagnostic criteria
Ventricular remodeling pathophysiology
Myocardial rupture risk factors
Sinoatrial arrest pathophysiology
Tuberculous pericarditis pathophysiology
Constrictive pericarditis risk factors