The pathophysiology of<unk>  pericarditis is not fully understood. However, there is a correlation observed with levels of blood urea nitrogen(usually >60 mg/dL) and creatinine. In renal failure, the absence or inadequate dialysis can lead to accumulation of these toxins in the body which may cause inflammation of pericardium and development of adhesions between the two pericardial layers. This could lead to loculation of effusion in pericardial cavity. Patients undergoing dialysis may also develop pericarditis. In a series, 13% of patients undergoing hemodialysis developed pericarditis.
Uremic pericarditis pathophysiology
Tuberculous pericarditis pathophysiology
HIV induced pericarditis pathophysiology
Endocarditis pathophysiology
Myocarditis pathophysiology
Papillary muscle rupture pathophysiology
Pericardial effusion epidemiology and demographics
Ventricular remodeling pathophysiology