S. schenckii is usually transmitted via posttraumatic inoculation to the human host, however, infrequently<unk>  may also develop as a result of spore inhalation. The pathophysiology of<unk>  depends on the histological subtype and the frequently nonspecific histopathology may mimic other granulomatous diseases. S. schenckii is capable of modulating the immune response to promote its own survival by blocking cytokine production by macrophages.
Sporotrichosis pathophysiology
Actinomycosis pathophysiology
Blastomycosis pathophysiology
Dermatophytosis pathophysiology
Coccidioidomycosis pathophysiology
Mucormycosis pathophysiology
Mycosis fungoides pathophysiology
Tularemia pathophysiology