<unk>   is caused by a loss of skin melanocytes.  Although the exact mechanism is not known, at least in some cases, an autoimmune process may play a role.   The fact that<unk>  is more prevalent in patients with certain autoimmune disorders, such as Addison's disease, hyperthyroidism, alopecia areata and pernicious anemia supports this hypothesis, but it should also be recognized that the majority of patients with<unk>  do not have any autoimmune disorder.
Vitiligo pathophysiology
Scleroderma pathophysiology
Dysplastic nevus pathophysiology
Squamous cell carcinoma of the skin pathophysiology
Psoriasis pathophysiology
Uveal melanoma pathophysiology
Scleroderma risk factors
Scleroderma laboratory findings