The pathogenesis of<unk><unk>  is directly connected to the pathogen responsible for nasopharyngitis. This includes infectious causes, such as viral upper respiratory infection, as well as bacterial infection from Streptococcus pneumoniae, Haemophilus influenzae, Moraxella catarrhalis, and Staphylococcus aureus. Mucus in the middle ear causes congestion that results in dysfunction of the eustachian tube. Pressure regulation in the inner ear is altered, causing effusion of fluid into the tympanic cavity containing the pathogen of nasopharyngitis.<unk><unk>  results from the inflammatory response to the infection.<unk><unk>  is transmitted through respiratory droplets through saliva or mucus, as well as direct physical contact with a contaminated individual or physical surface.<unk><unk>  is often associated with other upper respiratory conditions caused by the nasopharynx pathogen, as well as allergic conditions such as allergic rhinitis. There is evidence of genetic predisposition to<unk><unk> , with statistically significant evidence that it has high heritability. The following genes have been identified as having having potential pathogenic qualities for<unk><unk> : CAPN14, GALNT14, BPIFA3, BPIFA1, BMP5, GALNT13, NELL1, TGFB3. Up-regulation of the genes correlated to<unk><unk>  pathogenesis contribute to individual susecptibility to the disease.
Otitis media pathophysiology
Aortitis pathophysiology
Volvulus pathophysiology
Otitis externa pathophysiology
Rhinitis pathophysiology
Lymphangitis pathophysiology
Goiter pathophysiology
Gastritis pathophysiology