<unk>  may develop as a result of one of the three mechanisms: impaired granulocyte production, margination, and peripheral destruction. Genes involved in the pathogenesis of<unk>  include ELA2, HAX1, and CXCR4.
Neutropenia pathophysiology
Myelodysplastic syndrome pathophysiology
Thrombocytopenia pathophysiology
Eosinophilia pathophysiology
Allergy pathophysiology
Non-Hodgkin lymphoma pathophysiology
Neutropenia risk factors
Myelofibrosis pathophysiology