The major pathophysiologic mechanism responsible for the clinical manifestation of hepatorenal<unk>  is renal vasoconstriction. The hemodynamic disturbances include increased cardiac output, systemic vasodilatation and low arterial blood pressure. Thus, renal vasoconstriction occurs even with a normal blood volume and increased cardiac output.
Hepatorenal syndrome pathophysiology
Renal papillary necrosis pathophysiology
Acute liver failure pathophysiology
Acute kidney injury pathophysiology
Hepatorenal syndrome risk factors
Ascites pathophysiology
Acute tubular necrosis pathophysiology
Hepatorenal syndrome causes