The exact pathogenesis of<unk>  thyroiditis is not fully understood. It is thought that<unk>  thyroiditis is the result of an autoimmune phenomenon. Activated matured T-cells (HLA-DR+CD3+), activated helper/inducer T-cells (HLA-DR+CD4+), activated suppressor, and cytotoxic T-cells (HLA-DR+CD8+) were higher in patients with<unk>  thyroiditis as compared to the healthy controls. It indicates that the activation of T cells, especially helper/inducer T cells, might have an important role in the pathogenesis of<unk>  thyroiditis.<unk>  thyroiditis is associated with the HLA-DR3 and DR5 genes. Lymphocytic infiltration of the thyroid gland, the absence of Hurthle cells and germinal centers on histological analysis are the microscopic histopathological findings suggestive of<unk>  thyroiditis.
Silent thyroiditis pathophysiology
Hashimoto's thyroiditis pathophysiology
De Quervain's thyroiditis pathophysiology
Postpartum thyroiditis pathophysiology
Thyroid adenoma pathophysiology
Medullary thyroid cancer pathophysiology
Thyroid nodule pathophysiology
Anaplastic thyroid cancer pathophysiology