<unk>  results from the activation of the body's inflammatory response to bacterial exposure. In the absence of an appropriate immune response to the initial bacterial invasion, the infection can spread systemically through the bloodstream. Certain risk factors predispose an individual to develop<unk>  which includes the breaking of the skin, previous unresolved skin infections, and immunosuppression.
Cellulitis pathophysiology
Gangrene pathophysiology
Erysipelas pathophysiology
Lymphangitis pathophysiology
Stevens-Johnson syndrome pathophysiology
Allergy pathophysiology
Cellulitis risk factors
Hand-foot-and-mouth disease pathophysiology