The pathogenesis of<unk>  abscess is still not well-understood. Some authorities have proposed that<unk>  abscess arises from blockage of drainage from tonsillar crypt following acute tonsillitis resulting in spread of infection into the<unk>  space. However, others believe infectious process involving Weber's gland located in the supratonsillar space account for the abscess formation. Antigenic response following any disturbance arising from within or around the tonsillar crypt mucosa allows for lymphocytic interaction. This disruption in the crypt epithelium may be preceded by infectious process. Invasion and proliferation of the tonsillar crypt by infectious pathogens results in localized edema and influx of neutrophils. This is clinically seen as inflamed tonsil with or without exudation. Pus accumulation within tissue behind the supratonsillar space leads to tonsillar bulging, uvula and palate deviation.
Peritonsillar abscess pathophysiology
Tonsillitis pathophysiology
Epiglottitis pathophysiology
Pharyngitis pathophysiology
Rhinitis pathophysiology
Peritonsillar abscess risk factors
Strep throat pathophysiology
Bronchogenic cyst pathophysiology