<unk> , also known as testicular feminization, is an x-linked recessive condition resulting in a failure of normal masculinization of the external genitalia. Mutations in the<unk>  receptor (AR) gene is the cause of this disease. Depending on the amount of residual receptor function,<unk>  can be divided into two categories: complete<unk>  and incomplete<unk> . The typical characteristic of this disease is feminization. The tests in blood levels of hormones, such as testosterone, dihydrotestosterone (DHT), follicle-stimulating hormone (FSH) and luteinizing hormone (LH) may be helpful for the diagnosis. 
Treatments may be complex, invloving medical practice and medical ethics. The options are hormone replacement therapy, surgery and psychosocial support. For patients with complete<unk> , prognosis is good after orchidectomy at the proper time. Prognosis of incomplete<unk>  patients depends on the presence and severity of ambiguous genitalia.
Androgen insensitivity syndrome
Kallmann syndrome
Kallman syndrome
Turner syndrome
Wolman disease
Kostmann syndrome
Angelman syndrome
Hypogonadotrophic hypogonadism