Bloodstream infections<unk>  by endogenous flora and reactivation of latent infections account for a majority of initial<unk>  episode in neutropenic patients with cancer.  Bacterial isolates commonly<unk> bacteremia in the setting of neutropenia followed by fungi and viruses..  Certain endogenous microorganisms may be reactivated and exit latency during immunosuppression. These include herpes simplex virus, varicella-zoster virus, Epstein-Barr virus, cytomegalovirus, hepatitis B and C viruses, and Mycobacterium tuberculosis.  Exogenous pathogens carried by contaminated blood products, medical equipment and devices, water sources, and health care workers represent less common sources of infection. These include Clostridium difficile, respiratory syncytial virus, vancomycin-resistant enterococci, and other multidrug resistant bacteria..  Fungal infections often take place in the setting of prolonged or profound neutropenia after administration of empirical therapy.  Candidiasis may range in severity from mucosal or cutaneous infection to septicemia, endocarditis, or disseminated infection.  Aspergillus, on the contrary, typically<unk>  life-threatening infection of the sinuses and lungs, particularly after protracted neutropenia.
Febrile neutropenia causes
Chronic myelogenous leukemia causes
Acute myeloid leukemia causes
Lymphocytopenia causes
Leukocytosis causes
Myeloproliferative neoplasm causes
Myelofibrosis causes
Febrile neutropenia risk factors