<unk>  or<unk> , also called mesangiocapillary glomerulonephritis is a type of glomerulonephritis caused by immune complexes depositing in the kidney glomerular mesangium and basement membrane (GBM), these immune complexes activate complement resulting in damaging the glomeruli.  The GBM is rebuilt ontop of the deposits, and shows "tram-tracking" appearance under the microscope.<unk>  <unk>  is a relatively uncommon inflammatory glomerulopathy that can cause chronic nephritis. Based on the histological pattern of glomerular injury it has been described as a chronic kidney disease found mostly in children and young adults. Like many forms of glomerulopathies,<unk>  (glomerulopathy) has been a diagnosis of tissue pathology rather the diagnosis of a specific disease entity. Therefore, the term membranoploriferative glomerulonephritis <unk>  relates to a pattern of glomerular injury characterized by mesangial proliferation and expansion, lobularization of the glomerular tufts and double contours which can be caused by many disease states. Glomerular injury occurs due to deposition of immune complexes on the glomerular mesangium or on the glomerular basement membrane. <unk>  has been categorized into 3 types based on the basis of histological pattern of glomerular damage. Clinically,<unk>  often present with hematuria, varying degrees of proteinuria, with or without Glomerular filtration rate impairment depending on the severity of glomerular injury, and the underlying etiology. The treatment of<unk>  <unk>  needs to address the underlying cause of the<unk> , eg, infection. The factors that predict renal prognosis should be assessed, and finally immunosuppressive drugs can be used when the underlying cause is autoimmune diseases.
Membranoproliferative glomerulonephritis
Membranous glomerulonephritis
Mesangial proliferative glomerulonephritis
Endocapillary proliferative glomerulonephritis
Rapidly progressive glomerulonephritis
Membranous nephritis
IgA nephropathy
C3 glomerulopathy