Development of<unk><unk>  is the result of genetic mutations, overexpression of angiogenic fators and downregulation of inhibitors of angiogenesis.<unk><unk>  may be associated with Kasabach-Merritt syndrome, hereditary hemorrhagic telangiectasia,<unk>  arterio-portal shunts, extra<unk><unk> ta, hemolytic anemia, focal nodular hyperplasia. On gross pathology, variable in size, well circumscribed, and classically subcapsular are findings of<unk><unk> . On microscopic histopathological analysis channels lined by benign endothelium containing RBCs, surrounding (non-endothelial) cells without significant atypia are findings of<unk><unk> .
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