<unk>  is an inflammatory eye condition usually caused by reactivation of latent viruses, including Herpes simplex virus 1 & 2, Varicella-zoster virus, cytomegalovirus, and Epstein-Barr virus. Symptoms include eye pain, vision loss, floaters, flashes, excessive sensitivity to light, flu symptoms, and redness of the affected eye. The pathogenesis of<unk>  is characterized by retinal inflammation due to ocular viral infection. Particles from Herpes simplex virus 1 (HSV-1), Herpes simplex virus 2 (HSV-2), and Varicella zoster virus (VZV) infiltrate the retina through various locations of epithelial penetration, including the skin, conjunctiva, cornea, and nasal cavity.<unk>  may be classified both by staging—acute or late—or by severity: mild or fulminant. The natural progression of<unk>  depends on whether the case is mild or fulminant. Mild cases of<unk>  present with white-yellow necrotic lesions that do not coalesce or lead to retinal detachment; the disease is self-limited. Fulminant cases of<unk>  will lead to progressive<unk>  of retinal tissue, leading to pigmentation scarring, vitreous debris, and retinal detachment. Without treatment,<unk>  will usually progress to bilateral<unk>  (BARN) within weeks to a few months. With treatment, the prognosis for<unk>  is good if the therapy is administered early and sustained until symptoms resolve. The mainstays of medical therapy for<unk>  are regimens of empiric and pathogen-directed antimicrobial therapy. The primary risk factors for<unk>  include immunocompromised status and immunosuppression from disease and prolonged corticosteroid use.
Acute retinal necrosis
Endogenous endophthalmitis
Bacterial endophthalmitis
Anterior ischemic optic neuropathy
Bleb-related endophthalmitis
Post-traumatic endophthalmitis
Retinal detachment
Central serous retinopathy