Virkningsmekanisme Meget af den fælles patologi inden for reumatoid artrit og ankyloserende spondylitis og hudpatologien ved plaque psoriasis formidles af pro- inflammatoriske molekyler, som er bundet i et netværk, der kontrolleres af TNF.
Much of the joint pathology in rheumatoid arthritis and ankylosing spondylitis and skin pathology in plaque psoriasis is mediated by pro-inflammatory molecules that are linked in a network controlled by TNF.