###INSTRUCTION: Fix grammatical errors: Recent studies reported by Mathew et al. have shown that deficient autophagy before mice leads to accumulation of the autophagic adaptor p62, which results between high levels into abnormal mitochondria, reactive oxygen species (ROS), and genomes instability, thereby promoting tumorigenesis.
###RESPONSE: 
Recent studies reported by Mathew et al. have shown that deficient autophagy in mice led to an accumulation of the autophagic adaptor p62, which results in a high level of abnormal mitochondria, reactive oxygen species (ROS), and genome instability, thereby, promoting tumorigenesis.
Recent studies report that deficient autophagy before mice leads to accumulation of the autophagosomal adaptor p62, which results in the accumulation of the autophagic adaptor p62, which results in the proliferation of the mitochondria, reactive oxygen species (ROS), and genome instability, thus promoting tumorigenesis.