Snake venom phospholipase A2 homolog that lacks enzymatic activity. In vivo, it displays local myotoxin and edema-inducing activities and is lethal by intraperitoneal injection. The myotoxicity effect is weaker in comparison to other myotoxins, probably due to the formation of high molecular weight complexes and to the oligomeric conformation (conventional dimer). It shows specificity toward neurons and myotubes, but not on a variety of other cell types. This PLA2 excites a cohort of sensory neurons via ATP release and consequent activation of P2RX2 and/or P2RX3 purinergic receptors. Pannexin hemichannels act as downstream mediators of toxin-evoked ATP release. In vivo, it elicits nonneurogenic inflammatory pain, thermal hyperalgesia, and mechanical allodynia, of which the latter is completely dependent on purinergic signaling