hat
Si yon tretman dwòg nan san presyon ki nesesè, sa yo rele beta-blockers yo te itilize pami lòt bagay. Non yo derive beta-blockers yo soti nan beta-reseptè yo. Yon moun ka byen imajine sa yo reseptè kòm "pwen debakadè" pou òmòn ak lòt "sibstans mesaje" sou selil yo. Yo travay selon prensip kle-fèmen: Chak reseptè se yon seri ki ka klete ak sèten "kle", sètadi òmòn sèten. Epinephrine nerotransmetrite yo ak norepinephrine, ki fè yo de pli zan pli jwenn nan kò a pandan estrès, ogmante san presyon atravè de mekanis:
eng
If a drug treatment of high blood pressure is necessary, so-called beta-blockers are used among other things. Their name derives the beta-blockers from the beta-receptors. One can well imagine these receptors as "docking points" for hormones and other "messenger substances" on the cells. They work according to the key-lock principle: Each receptor is a lock that can be unlocked with certain "keys", ie certain hormones. The neurotransmitters epinephrine and norepinephrine, which are increasingly found in the body during stress, increase blood pressure via two mechanisms: