hat
Pulmonèr - Ekspansyon an oksijèn ak aparèy newolojik yo souvan site kòm enkyetid enpòtan avèk HBOT. Oksijèn tolerans Oksijèn ki evite manifestasyon sa yo byen defini pou ekspoze kontinyèl nan moun nòmal. Sentòm lenmonter yo pa pwodwi pa ekspozisyon chak jou nan oksijèn nan 2.0 oswa 2.4 ATA pou èdtan 2 oswa 1.5. Ensidans la nan convulsion oksijèn lè w ap itilize menm jan an ekspoze se sou 1 pou chak tretman pasyan 10,000. Menm lè reyaksyon oksijèn rive, pa gen okenn efè rezidyèl si yo kapab evite chòk mekanik. Pasyan ki gen blokaj ayewopò gen yon risk ogmante pou balotrauma poumon pandan dekonpwesyon. Lonbraj barotrauma pandan dekonpwesyon se bagay ki ra.
eng
Pulmonary – Pulmonary and neurological manifestations of oxygen poisoning are often cited as major concerns with HBOT. Oxygen tolerance limits that avoid these manifestations are well defined for continuous exposure in normal people. Pulmonary symptoms are not produced by daily exposure to oxygen at 2.0 or 2.4 ATA for 2 or 1.5 hours respectively. The incidence of oxygen convulsions when using similar exposure is about 1 per 10,000 patient treatments. Even when oxygen convulsions do occur, there are no residual effects if mechanical trauma can be avoided. Patients with airway obstruction have an increased risk for pulmonary barotrauma during decompression. Pulmonary barotrauma during decompression is rare.