hat
Chèchè yo demontre ke pa redwi aktivite a nan ATP synthase, yo te kapab pwoteje selil neronal soti nan yon kantite toksisite ki asosye ak aje nan sèvo an. Youn nan rezon pou efè neroprotective sa a panse yo dwe wòl nan excitotoxicity nan domaj selil newòn.
eng
The researchers demonstrated that by reducing the activity of ATP synthase, they were able to protect neuronal cells from a number of toxicities associated with the aging of the brain. One reason for this neuroprotective effect is thought to be the role of excitotoxicity in neuronal cell damage.