hat
Pi gwo efè segondè Andriol yo enkli efè segondè androgenic yo. Sa yo se masculinizing ak gason seksyèl segondè seksyon karakteristik ki gen rapò ak lòt. Pou ou kab vin ke Andriol se Testostewòn, ak Testostewòn se prensipal android gason an, li gen enfliyans dirèk sou efè segondè Androjenik. Anplis de sa, segondè Androjenik la nan pwodwi testostewòn se plis atribuabl rediksyon li yo (oswa konvèsyon) nan Dihydrotestosterone (DHT), ki se yon androjèn byen lwen plis ki pagen pase Testostewòn tèt li. Nan ka sa a, itilizasyon yon inhibite reduksyon 5-Alpha ka bese efè segondè androgenic pa fason pou anpeche oswa diminye konvèsyon nan Testostewòn nan DHT. Efè segondè andojèn yo enkli: ogmantasyon nan agresyon, ogmante kwasans cheve nan kò, ogmante sekresyon po lwil, grandisan nan vwa a, ak posib chod modèl gason (MPB) nan moun sa yo ki gen jenetikman predispoze li.
eng
The next major Andriol side effects include androgenic side effects. These are masculinizing and male secondary sex characteristic related side effects. Being that Andriol is Testosterone, and Testosterone is the primary male androgen, it has direct influence on androgenic side effects. Furthermore, the high androgenicity of Testosterone products is more attributable to its reduction (or conversion) into Dihydrotestosterone (DHT), which is a far more potent androgen than Testosterone itself. In this case, the use of a 5-alpha reductase inhibitor can mitigate androgenic side effects by way of preventing or reducing the conversion of Testosterone into DHT. Androgenic side effects include: increases in aggression, increased body hair growth, increased oily skin secretions, deepening of the voice, and possible male pattern baldness (MPB) in those men that are genetically predisposed to it.