hat
Clenbuterol ka kòmanse reyaksyon fizyolojik menm jan ak adrenalin. Li aktive subtip yo reseptè beta-2 nan yon sistèm adrenèrjik. Sa rezilta nan ogmantasyon nan CAMP (Derivatif ATP ki nesesè pou selil kwa-pale). Kòm yon rezilta, pwoteyin Kinase A ogmante. Pwoteyin kinase A se yon anzim ki kontwole aktivite lipid, glikojèn, ak sik metabolis. Ogmantasyon li stimul yon efè boule grès nan kò a. Li amelyore lòt chemen ki ede nan boule grès atravè yon pwosesis byolojik li te ye tankou oksidasyon asid gra.
eng
Clenbuterol can initiate physiological reactions similar to adrenaline. It activates the beta-2 receptor subtypes in an adrenergic system. This results in the increment of cAMP (ATP’s derivative needed for cellular cross-talk). As a result, protein kinase A increases. Protein kinase A is an enzyme which regulates lipid, glycogen, and sugar metabolism activity. Its increase stimulates a fat burning effect in the body. It enhances other pathways which assist in burning fat via a biological process known as fatty acid oxidation.