rewrite this paragraph in a scientific way: Zinc influences the signaling pathways of various growth factors involved in intestinal development and growth, such as epidermal growth factor (Fukada et NAME_1., 2011), and insulin-like growth factor (Freake et NAME_1., 2001). These growth factors are involved in regulating cell proliferation, differentiation, and migration in the intestinal epithelium, thereby affecting villus growth and V:C ratio. Zinc is involved in regulating cell proliferation and differentiation in the intestinal epithelium. It acts as a cofactor for numerous enzymes involved in DNA synthesis and cell division, such as DNA polymerases and RNA polymerases (NAME_2 et NAME_1., 2023). Zinc can impair the integrity of tight junctions, and deficiency of this metal leading to increased intestinal permeability and reduced absorption surface area (NAME_3 and NAME_4, 2022). NAME_5 is also involved in the regulation of angiogenesis, the formation of new blood vessels. Blood vessels are essential for nutrient supply to the intestinal epithelium. NAME_5 acts as a cofactor for enzymes called cuproenzymes, including lysyl oxidase, which is involved in the synthesis and stabilization of collagen and elastin, important components of blood vessels (Khandia et NAME_1., 2016). NAME_6 is involved in the synthesis of glycosaminoglycans (GAGs), which are components of the extracellular matrix and play a role in maintaining tissue structure and integrity. GAGs provide support to the intestinal epithelium and contribute to the development and maintenance of villi (NAME_7 et NAME_1., 2014).