Speaker 1: There are three possible ways for
Lyme disease to be transmitted.
Number one: By the bite of an insect or tick
that transfers blood. Two: In a pregnant woman
from the mother to the fetus via the placenta.
Three: Sexually transmitted. Lyme also contains
coinfections such as Babesia, Ehrlichia, Bartonella
and secondary infections such as Mycoplasma,
Candida, and viruses as HHV-6, cytomegalovirus,
and Epstein-Barr.
Lyme is characterized by a spirochete infection.
The spirochete releases bacterial lipoproteins,
that is a neurotoxin. The BLP neurotoxins
are associated with symptoms like memory problems,
burning neurological pain, and numbness. The
spirochete moves in a corkscrew-like motion
as it burrows its way through the tissue and
organs of the body. The spirochete contains
antigens and the antigens are like fingerprints,
which identify this infection to the body's
immune system.
The dendritic cell is a key part of the immune
system. One of the major functions of the
dendritic cell is to process the antigen and
present it to other parts of the immune system.
This is why the dendritic cell is called the
antigen-presenting cell.
Once the spirochete enters the human body,
it comes into contact with the dendritic cell
of the immune system. The spirochete rubs
or displaces the antigens onto the dendritic
cell. Then the dendritic cell begins to process
the antigen of Lyme disease for the other
parts of the immune system to recognize it.
The helper T cells come along and pick up
the processed antigen off of the dendritic
cell. Then the helper T cells proceed to pass
the antigens along to another portion of the
immune system called the killer T cells. The
job of the killer T cell is to hunt down the
spirochete infection and eliminate it by using
the antigens to identify the Lyme disease.
As the spirochete burrows into the body, it
continues to spread its antigen onto the surface
of healthy tissue and organs. The killer T
cells follow as they seek out the antigen
of the spirochete. In this process, the killer
T cells begin to destroy healthy tissues and
organs because they cannot differentiate between
the antigen of the spirochete or the antigen
of healthy tissue.
As the killer T cells come into contact with
the antigen located on the healthy tissue,
we begin to see inflammation and destruction
of the healthy tissue. This is clearly seen
in autoimmune disease process where the immune
system cannot distinguish between self and
nonself. As the spirochete is continuously
moving, it's releasing the BLPs, the bacterial
lipoprotein which is impairing the immune
system and causing inflammation, irritation,
and damage to the peripheral and central nervous
systems.
The problem remains the number of cases of
Lyme disease reported to the CDC do not represent
the total number of cases that exist. Lyme
disease, its coinfections and secondary infections
may represent more than 300 different medical
conditions such as chronic fatigue, fibromyalgia
and other autoimmune diseases.
The following diagram is designed to help
explain the various scenarios of chronic Lyme
disease treatment. There are three major focal
points in the treatment of Lyme disease. One:
The disease itself, which is represented by
Lyme, its coinfections, and secondary infections.
Two: The immune system such as CD-57 and other
immune modulators. Three: The neurotoxins
or the bacterial lipoproteins in yellow that
usually take a longer time to clear up as
the infection improves.
As the immune system increases and the infection
decreases over time, which is represented
in the yellow line, we see the windows of
symptoms improving. Also notice that it takes
the bacterial lipoprotein neurotoxins longer
to clear. This represents the ideal course
of treatment. Unfortunately, this does not
occur in the current methods of treatment.
Now let's review three common treatment scenarios.
Number one: The patient is diagnosed with
Lyme disease and then given four to six weeks
of IV antibiotics. You'll notice it does nothing
for the immune system except impair immune
function slightly shifting the disease over
four to six weeks. Then you'll see the relapse
once the antibiotic treatment ends. You'll
notice the antibiotics do nothing to address
the coinfections, the secondary infections,
or the neurotoxins related to the Lyme disease
and it does nothing to strengthen the immune
system.
In the second scenario, you'll see the patient
is using IV antibiotics, dietary herbs and
supplements. The IV antibiotics bring the
infection down a little bit and the dietary
herbs and supplements slightly increase the
immune system. As in the first example, once
the patient stops therapy we see another relapse.
The third scenario represents a patient using
advanced immunotherapy. Notice the sharp increase
and rise in the immune system. In addition
to the IV antibiotics used to treat the infection,
you'll see aggressive natural antiviral, antifungal
treatments to fight the infection, and you'll
notice a sharp decrease in the elimination
of the infection. Finally notice biodetoxification
that is used to help the neurotoxin and the
bacterial lipoproteins to be facilitated out
of the body. This represents the Envita advantage
in utilizing a comprehensive treatment plan
to treat Lyme disease.
